NUR 57500

Pathophysiologic Concepts For Advanced Practice Nursing II

Purdue University Northwest · UGRD · Fall 2026

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Students examine the principles of physiologic and pathophysiologic function at all levels of organization from cells to organs, systems and the human organism. They apply these physiologic and pathophysiologic concepts to understand how illness and disease alter cell and organ/system function that lead to clinical manifestations. The course uses homeostasis as a model to account for regulatory and compensatory functions in health and illness across the lifespan. Students develop the necessary theoretical and empirical foundation of subsequent understanding of the diagnosis and management of integrated human responses to disease and aging. This course is one of a two-semester series; this term concepts covered include disorders of blood and lymph, cardiovascular pulmonary function and dysfunction, reproductive health and disease, hormone regulation and endocrine disease, nutrition and elimination, and renal function and dysfunction. Typically offered Fall. Course Learning Outcomes 1. Apply the model of homeostasis to the understanding of the theoretical/empirical basis of physiologic regulation and pathophysiologic phenomena of the human body (Aligns with program outcome 1). (Application) 2. Describe abnormal physiological disorders in the human population. (Aligns with Program Outcome 1). (Knowledge) 3. Examine the interaction between genetics and environmental factors in the development of a given disorder (Aligns with Program Outcomes 1, 2). (Comprehension) 4. Distinguish the signs, symptoms, and diagnostic test results associated with disorders affecting the human population (Aligns with Program Outcomes 1, 3) (Analysis) 5. Integrate the cultural, socioeconomic, gender, and age-related differences in the pathophysiologic response and treatment associated to disease or injury. (Aligns to program outcomes 2, 4). (Synthesis) View Class Schedule

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Class #purdue_northwest-2026Fall 2026UGRD3.00 credits
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